acidosis

Shock , sepsis, acidosis , hypoxia, and so forth, are treated.

对休克,感染, 酸中毒, 缺氧等应加以治疗.

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Chronic respiratory alkalosis ( CRA ) , CRA plus metabolic alkalosis and metabolic acidosis are seldom.

慢性呼吸性碱中毒 、 性呼吸性碱中毒并代谢性碱中毒和代谢性酸中毒是少见的.

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A number of intoxicants are associated with metabolic acidosis.

许多毒性物质可引起代谢性酸中毒.

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Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血液学研究显示有严重高磷血症 、 低钙血症和代谢性酸中毒.

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Results CVVH, HDF and HD showed definite effects on severe lactic acidosis.

结果CVVH 、 hdF和hd治疗乳酸酸中毒疗效明确.

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Methods: The animals achieved hypercapnic acidosis with mechanical hypoventilation before ischemia - reperfusion.

方法在缺血再灌注前,用低潮气量,低频率的机械通气方法造成动物的呼吸性酸中毒.

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All patients presented with metabolic abnormalities, including hypoglycemia, and lactic acidosis.

所有这些病人都表现出代谢异常, 包括低血糖血症和乳酸性酸中毒.

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Metabolic acidosis was resolved by both hemoglobin solutions, but not by dextran.

两组均无代谢性酸中毒, 而右旋糖酐组有.

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Retinal NV induced by acidosis relates to VEGF.

酸中毒引起的视网膜新生血管与VEGF有关.

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Methanol can cause poisoning, systemic acidosis, optic nerve damage and central nervous system ( CNS ) effects.

甲醇能造成中毒, 组织酸毒症, 选择性神经伤害和影响中枢神经.

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Hypercalciuria was considered as a secondary condition when ( It was ) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒,就可以认为是继发病.

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Respiratory acidosis is caused by retention of CO 2 secondary to decreased alveolar ventilation.

呼吸性酸中毒是由继发于肺泡通气功能降低引起的CO2贮留所致.

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Biochemical investigations showed hyponatremia , hyperkalemia, hypochloremia, metabolic acidosis and large amount of urinary sodium excretion.

经过适当治疗和水分补充,临床肠胃症状很快获得缓解.

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Risk factors for AGML low blood pressure , damage of liver function, serious infection, acidosis and uremia.

临床分析提示,低血压 、 功能损害 、 重感染 、 中毒、毒症等是急性胃粘膜病变的危险因素.

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RESULTS: Hemorrhagic shock reduced functional capillary density ( FCD ) by 70 % and caused significant metabolic acidosis.

结果: 失血性休克使毛细血管密度 ( FCD ) 减少70%,引起显著的代谢性酸中毒.

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